Neurochemical Patterns in Patients with Post-Vaccination Neurological Symptoms (Pfizer mRNA & Gardasil)
- Gary Moller
- Jul 4
- 5 min read
CLINICAL COMPARATIVE REPORT
Notice: To preserve patient privacy, all names and identifying details in this report have been changed or omitted. Pseudonyms (first names only) have been used throughout.
1. Clinical Objective
This report presents a comparative analysis of seven patients who developed a range of neurological and systemic symptoms following administration of either the Pfizer mRNA COVID-19 vaccine or the Gardasil HPV vaccine. Each patient underwent comprehensive biochemical testing, including advanced neurotransmitter and inflammatory profiling.
The core aim of this report is to:
Identify shared neurochemical and mitochondrial patterns across cases
Understand how these patterns align with reported symptoms
Establish a medically and legally robust link between the vaccine and injury
Evaluate these findings against the standards used by ACC and under commercial insurance law
2. Clinical Context – Symptom Range and Rationale for Testing
The symptoms reported by patients varied in presentation and intensity but clearly shared underlying features of neuroinflammation and mitochondrial dysfunction. These two mechanisms are foundational to the functioning of the central and peripheral nervous systems, making their disruption deeply debilitating.
Reported symptoms in these patients included:
Cardiovascular irregularities, icluding myocarditis
Varicose veins, deep vein thrombosis
Tremors, muscle spasms
Inability to speak or swallow
Partial or complete loss of control of limbs
Paralysis
Extreme, unrelenting fatigue
Musculoskeletal injuries, tendons, ligaments, that fail to heal.
Easy bruising.
Loss of smell and taste
Impaired balance and coordination
Visual disturbances
Tinnitus
Severe anxiety, depression, panic attacks
Bizarre, antisocial, or erratic behaviour
Thankfully, other conditions, including stroke and cancer are not reported in this small cohort of patients.
Some symptoms were initially mild and transitory; however, in most cases, they worsened over time, leading to significant suffering and disability. It is common in post-vaccine injuries for patients to delay seeking help, thinking the symptoms will resolve naturally. This delay should not disqualify or diminish the seriousness of these claims.
Based on this symptomatology, comprehensive testing was initiated. Neurotransmitter panels and mitochondrial markers were selected to confirm clinical suspicion of central nervous system injury and cellular energy collapse.
Take note: None of the neurologists, or cardiologists, nor the ACC, ordered this kind of testing. Is this a case of covering one's eyes, then declaring with authority, that there is nothing to see? Case closed!
3. Common Biochemical Patterns
Despite symptom differences, all seven patients demonstrated a highly consistent biochemical profile:
a. Neuroinflammation – Tryptophan Diversion
In nearly all cases, tryptophan metabolism was abnormally shifted from serotonin production toward the kynurenine pathway, producing neurotoxic metabolites such as kynurenic and xanthurenic acid. This strongly suggests chronic neuroinflammation and immune activation, a common finding in vaccine-associated injuries.
b. Excitatory Overload and Poor Inhibition
Elevated glutamate (a primary excitatory neurotransmitter) and depressed or overcompensated GABA (primary inhibitor) reflected a state of neuronal excitotoxicity. These imbalances cause hyperstimulation, anxiety, insomnia, and can accelerate neuronal injury when left unresolved.
c. Catecholamine Dysregulation
Catecholamines (dopamine, noradrenaline, adrenaline) showed signs of initial overdrive in some cases (e.g., high noradrenaline in Anna), and subsequent collapse in others (e.g., dopamine/adrenaline depletion in Linda and Jack). This biphasic pattern reflects the transition from acute stress to adrenal burnout, often mediated by mitochondrial exhaustion.
d. Serotonin Breakdown
Patients like Anna and Maya had low 5-HIAA (serotonin metabolite), suggesting impaired serotonin synthesis or activity. Others, such as Rachel, showed elevated 5-HIAA, indicating serotonin is being metabolised too quickly – often due to inflammation-driven monoamine oxidase (MAO) activation.
4. Mitochondrial Dysfunction as the Root Cause
The biochemical disruption observed is rooted in mitochondrial failure, which impairs ATP production, redox balance, neurotransmitter recycling, and neuroprotection. This is a foundational collapse of cellular function.
Key mitochondrial roles affected:
Production of cellular energy (ATP)
Regulation of calcium and synaptic signalling
Recycling of glutamate and GABA
Detoxification of reactive oxygen species (ROS)
Maintenance of neural membrane potentials
Disruption of these processes leads to many of the observed symptoms: cognitive impairment, fatigue, anxiety, mood swings, autonomic dysfunction, muscle weakness, and sensory disturbances.
Objective tests confirmed mitochondrial distress in Anna, Rachel, Maya, and Helen, providing strong biomedical evidence supporting the observed neurochemical patterns and physical symptoms.
5. Functional Staging of Neurochemical Collapse
Stage | Features | Representative Patients |
Stage 1 – Hyperreactivity | High glutamate/PEA, low GABA, high catecholamines | Anna, Ella |
Stage 2 – Exhaustion | Low serotonin, rising GABA, adrenal instability | Maya, Helen |
Stage 3 – Suppression | Flattened dopamine/adrenaline, high inflammation, serotonin breakdown | Rachel, Linda, Jack |
These stages reflect a predictable trajectory from initial immune overstimulation, through metabolic compensation, to eventual collapse.
6. Shared Vaccine Components and the Bolus Theory
A common thread between these vaccine formulations is the inclusion of substances known to irritate and inflame the immune system. These include:
The synthetic spike protein instructions (mRNA vaccines)
Aluminium adjuvants (used in Gardasil)
Polyethylene glycol (PEG)Â and other excipients
These substances are not inherently benign. When delivered by injection as a concentrated bolus, they bypass many of the body's natural filters, such as mucosal and hepatic detoxification systems.
The Bolus Theory, as articulated by Marc Girardot, proposes that a concentrated introduction of immune irritants may overwhelm and confuse the immune system. This can lead to unintended consequences including systemic inflammation, autoimmunity, neurological misfiring, and long-term immune dysregulation.
This theory aligns closely with what is being observed in these patients. A single dose—or multiple cumulative doses—of these vaccines may act as a bolus insult, triggering cascading mitochondrial collapse, neuroinflammation, and multi-system symptoms.
7. Finishing Observations
These patients display a repeatable, medically coherent injury pattern following vaccination, consistent with:
Neuroimmune dysregulation
Central nervous system inflammation
Mitochondrial collapse
Neurotransmitter failure
These injuries are not coincidental or psychosomatic. They are measurable, replicable, and explainable within existing biomedical science.
8. Mitochondrial Dysfunction – Legal Weight and Objective Evidence
Of the four patients tested for mitochondria health, all had positive results indicating mitochondrial dysfunction, aligning with symptoms and neurotransmitter patterns. This adds substantial evidentiary weight.
Why mitochondria matter legally:
Mitochondrial damage provides a mechanism explaining symptom onset.
It is objectively measurable (via biochemical markers).
It aligns with temporal onset shortly after vaccination.
It offers causation clarity without requiring proof of fault.
Under commercial insurance law and ACC guidelines, a claim is valid when:
There is a personal injury (physical or psychological)
It was caused by a medical treatment (e.g. vaccination)
It was not an ordinary consequence of that treatment
The injury occurred despite proper administration (no fault required)
All of these cases meet these tests. The "balance of probabilities" standard – used in commercial law – is clearly satisfied by:
The tight temporal link to vaccination
The presence of objective mitochondrial dysfunction
The consistency of neurochemical injury across cases
Delays in seeking help, common in these cases, are medically understandable and do not disqualify claims, particularly when symptoms worsened over time and are now clearly disabling.
9. Conclusion
These cases form a medically and legally sound basis for ACC treatment injury claims. The evidence of mitochondrial collapse and neuroinflammation is clear and consistent across:
Symptom patterns
Neurotransmitter profiles
Functional staging
Objective test results
These injuries are real, serious, and disabling. ACC should accept these claims without delay, and clinicians should be encouraged to report and investigate similar patterns in other patients presenting post-vaccination neurological complaints.
Each case meets the legal, medical, and ethical thresholds for cover.